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Cold urticaria is a chronic, reactive skin disorder. It is probably the most common form of physical urticaria hives. Major symptoms may include abnormal reddening of the skin erythemahives and itching after exposure of the skin to cold temperatures. There are two forms of the disorder: essential acquired cold urticaria, and familial hereditary cold urticaria. The symptoms of the acquired form become obvious in two to five minutes after exposure to the triggering substance or situation, while it takes 24 to 48 hours for symptoms of familial cold urticaria to appear.

Also, symptoms tend to last longer with the familial form, typically about 24 hours although they may remain for as long as 48 hours. With the acquired form, symptoms tend to last for one to two hours. In cold urticaria, the skin has an abnormal reaction to cold. This may, for instance, occur after exposure Alberta cold weather or to swimming in cold water.

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The skin usually turns red, and develops welts and itching. This may be accompanied by fever, headache, anxiety, tiredness, and, sometimes, even fainting.

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Some persons may also have palpitations or wheezing. Symptoms Alberta familial cold urticaria may begin to appear as soon as 30 minutes after exposure to cold. They may persist for up to 48 hours after exposure. The redness and itching of the skin may be accompanied by fever, headache, tiredness, pain in the ts arthralgiaand the presence of excessive white blood cells leucocytosis in the blood.

Essential acquired cold urticaria consists, according to some clinicians, of several sub- such as primary acquired cold urticaria, delayed cold urticaria, localized cold urticaria, reflex cold urticaria or secondary cold urticaria, which are explained below:. Primary acquired cold urticaria can occur five to 30 minutes after exposure to cold. The reaction may occur in the cold itself, but more often during the rewarming phase. Itching and reddening of the skin may develop first, followed by a burning sensation.

Hives appear, usually lasting 30 minutes. The affected person may also experience headache, palpitations, wheezing or fainting.

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Localized cold urticaria has been reported to occur after exposure to cold at the sites of ragweed injections for allergies or ladybug bites. Reflex cold urticaria is characterized by widespread appearance of welts occurring in response to a drop in body temperature after localized exposure to cold applications e. Secondary cold urticaria can occur in connection with various blood disorders associated with viral infections such as mononucleosis. Cold urticaria can occur for unknown idiopathic reasons, or it may be transmitted as an autosomal dominant trait.

Human traits including the classic genetic diseases, are the product of the interaction of two genes for that condition, one received from the father and one from the mother. The risk of transmitting the disorder from affected parent to offspring is 50 percent for each pregnancy regardless of the sex of the resulting. The risk is the same for each pregnancy.

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The familial form of cold urticaria has been traced to the long arm of chromosome one 1q Chromosomes are found in the nucleus of all body cells. They carry the genetic characteristics of each individual. Pairs of human chromosomes are ed from 1 through 22, with an unequal 23rd pair of X and Y chromosomes for males and two X chromosomes for females. Chromosomes are further subdivided into bands that are ed.

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For example, chromosome 1q40 refers to band 40 on the long arm of chromosome 1. Some forms of cold urticaria are also diseases of the autoimmune system. Exposure of the skin to cold triggers symptoms of the disorder. Even such commonplace activities as walking in cold weather, swimming in cold water, or even bathing in cool water may trigger an episode. Cold urticaria affects males and females in equal s. Familial cold urticaria is rare.

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Symptoms of the following disorders can be similar to those of Cold Urticaria. Comparisons may be useful for a differential diagnosis:. It is characterized by spasms of blood vessels occurring especially in the fingers and toes. Intermittent attacks of pain, pallor or blue coloring cyanosis of the fingers or toes are precipitated by exposure to cold or by emotional upsets.

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The attacks last for minutes to hours, but are rarely severe enough to result in tissue loss. Rewarming the affected digits in normal blood circulation and a return to normal color and sensation. Onset usually occurs in the first or second decade of life. Cold Agglutination Disease is a blood disorder which occurs when the temperature of the blood is below body temperature.

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It is most pronounced below 25 C. Although it is seen occasionally in the blood of apparently healthy persons, it is more frequent in individuals with scarlet fever, staphylococcal infections, primary atypical pneumonia, certain hemolytic anemias, and trypanosomiasis. Paroxysmal Cold Hemoglobinuria is a disorder that makes the red blood corpuscles abnormally susceptible to antibodies which try to destroy them. It Cold feet at sex online Champion triggered by exposure to cold. Cold urticaria may be diagnosed by placing an ice cube on the skin of the subject's forearm for about four or five minutes.

A positive response is indicated by the appearance of a "hive" in the shape of the ice cube, within 10 minutes after the stimulus is removed. This two-step process suggests that exposure to cold is required, but hive formation actually occurs as the skin temperature rises. In some cases, cold-air exposure for 20 to 30 minutes is required to bring out the characteristic response. Treatment consists of patient education, stimulus avoidance and medication.

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The newer antihistamines H1 receptor blockers may be very effective. H2 receptor blockers such as Tegamet may also be effective. The induction of tolerance by repeated regional or generalized cold exposure has had variable. If attempted at all, such cold exposure should be performed on an in-patient basis and requires a well-motivated patient.

Treatment of cold urticaria may include the use of the drugs epinephrine, diphenhydramine, cyproheptadine, hydrochloride and cetirizine.

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Symptoms may be prevented with the use of warm clothing during cold weather. The avoidance of cold baths, swimming in cold water, etc. Information on current clinical trials is posted on the Internet at www. All studies receiving U. : [ protected]. Beers MH, Berkow R, eds. The Merck Manual, 17th ed. Berkow R, ed. The Merck Manual-Home Edition. Bennett JC, Plum F, eds.

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Cecil Textbook of Medicine. Philadelphia, PA: W. Saunders Co; Champion RH, et al. Textbook of Dermatology.

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Fire and ICE: the role of pyrin domain-containing proteins in inflammation and apoptosis. Clin Exp Rheumatol. New mutations of CIAS1 that are responsible for Muck;le-Wells synrome and familiar cold urticaria: a novel mutation underlies both syndromes. Am J Hum Genet. A case of cold-dependent exercise-induced anaphylaxis. Br J Dermatol. Mutation of a new gene encoding a putative pyrin-like protein causes familial cold autoinflammatory syndrome and Muckle-Wells syndrome. Nat Genet.

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Identification of a locus on chromosome 1q44 for familial cold urticaria. Cold stimulation test and histamine release in primary acquired cold urticaria. Int Arch Allergy Immunol. Cooke RA. Essential acquired cold urticaria: stimulated only by systemic as well as local cooling. Occup Med. Lanternier ML, Brannon K. Dermatology: Urticaria. In: Virtual Hospital. McKusick VA, ed. The content of the website and databases of the National Organization for Rare Disorders NORD is copyrighted and may not be reproduced, copied, downloaded or disseminated, in any way, for any commercial or public purpose, without prior written authorization and approval from NORD.

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If we don't have a program for you now, please continue to check back with us. General Discussion Cold urticaria is a chronic, reactive skin disorder. Essential acquired cold urticaria consists, according to some clinicians, of several sub- such as primary acquired cold urticaria, delayed cold urticaria, localized cold urticaria, reflex cold urticaria or secondary cold urticaria, which are explained below: Primary acquired cold urticaria can occur five to 30 minutes after exposure to cold.

Delayed cold urticaria may appear several hours after contact with the cold.

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